Pages that link to "Item:Q2632814"
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The following pages link to Model of influenza A virus infection: dynamics of viral antagonism and innate immune response (Q2632814):
Displaying 17 items.
- Dynamics of influenza virus and human host interactions during infection and replication cycle (Q371992) (← links)
- An in-host model of acute infection: measles as a case study (Q615413) (← links)
- A within-host mathematical model of H9N2 avian influenza infection and type-I interferon response pathways in chickens (Q827836) (← links)
- Mechanistic modeling explains the dsRNA length-dependent activation of the RIG-I mediated immune response (Q827851) (← links)
- Modeling the interaction of cytotoxic T lymphocytes and influenza virus infected epithelial cells (Q964170) (← links)
- Optimal control of acute myeloid leukaemia (Q1739304) (← links)
- A model for influenza with vaccination and antiviral treatment (Q1794908) (← links)
- Spatio-temporal dynamics of host-virus competition: a model study of influenza A (Q2010883) (← links)
- Modelling cross-reactivity and memory in the cellular adaptive immune response to influenza infection in the host (Q2013486) (← links)
- Compartmental model suggests importance of innate immune response to COVID-19 infection in rhesus macaques (Q2035797) (← links)
- Mathematical modelling of OAS2 activation by dsRNA and effects of dsRNA lengths (Q2133328) (← links)
- Age-related changes to the immune system exacerbate the inflammatory response to pandemic H1N1 infection (Q2154542) (← links)
- A dynamical model of human immune response to influenza A virus infection (Q2210022) (← links)
- Modelling within-host macrophage dynamics in influenza virus infection (Q2225936) (← links)
- Mathematical modelling of acute virus influenza A infections (Q2855792) (← links)
- Models of cytokine dynamics in the inflammatory response of viral zoonotic infectious diseases (Q5860542) (← links)
- A target-cell limited model can reproduce influenza infection dynamics in hosts with differing immune responses (Q6174302) (← links)