Pages that link to "Item:Q1705037"
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The following pages link to Interaction between synaptic inhibition and glial-potassium dynamics leads to diverse seizure transition modes in biophysical models of human focal seizures (Q1705037):
Displaying 15 items.
- The role of extracellular potassium dynamics in the different stages of ictal bursting and spreading depression: A computational study (Q1620798) (← links)
- Generalized seizures in a neural field model with bursting dynamics (Q1704935) (← links)
- Neural mass models as a tool to investigate neural dynamics during seizures (Q1705132) (← links)
- Extracellular potassium dynamics in the hyperexcitable state of the neuronal ictal activity (Q1948200) (← links)
- Interaction between thalamus and hippocampus in termination of amygdala-kindled seizures in mice (Q2011719) (← links)
- Transition dynamics and optogenetic control of epileptiform activity in a modified mean filed model of human cortex (Q2094434) (← links)
- A unified physiological framework of transitions between seizures, sustained ictal activity and depolarization block at the single neuron level (Q2163545) (← links)
- Interneuronal dynamics facilitate the initiation of spike block in cortical microcircuits (Q2163570) (← links)
- Role of potassium lateral diffusion in non-synaptic epilepsy: a computational study (Q2194967) (← links)
- Dynamics of a neuron-glia system: the occurrence of seizures and the influence of electroconvulsive stimuli. A mathematical and numerical study (Q2210981) (← links)
- Investigating the role of gap junctions in seizure wave propagation (Q2278564) (← links)
- Mitochondrial dysfunction and role in spreading depolarization and seizure (Q2281006) (← links)
- Inhibition modifies the effects of slow calcium-activated potassium channels on epileptiform activity in a neuronal network model (Q2500203) (← links)
- Idealized multiple-timescale model of cortical spreading depolarization initiation and pre-epileptic hyperexcitability caused by \(\mathrm{Na_V}1.1/ SCN1A\) mutations (Q6040846) (← links)
- Cell to network computational model of the epileptic human hippocampus suggests specific roles of network and channel dysfunctions in the ictal and interictal oscillations (Q6172449) (← links)
