Mathematical models of retinitis pigmentosa: the oxygen toxicity hypothesis
From MaRDI portal
Publication:1701586
DOI10.1016/j.jtbi.2017.05.006zbMath1381.92050OpenAlexW2610553401WikidataQ47854697 ScholiaQ47854697MaRDI QIDQ1701586
Philip J. Luthert, Paul A. Roberts, Eamonn A. Gaffney, Alexander J. E. Foss, Helen M. Byrne
Publication date: 27 February 2018
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: http://sro.sussex.ac.uk/id/eprint/82496/1/Roberts_et_al_2017_Final_Preprint.pdf
PDEs in connection with biology, chemistry and other natural sciences (35Q92) Medical applications (general) (92C50)
Related Items
Spatially-dependent model for rods and cones in the retina, The role of RdCVFL in a mathematical model of photoreceptor interactions, Mathematical models of retinitis pigmentosa: the trophic factor hypothesis
Cites Work
- Unnamed Item
- Photoreceptor death: spatiotemporal patterns arising from one-hit death kinetics and a diffusible cell death factor
- Optimal control in the treatment of retinitis pigmentosa
- Mathematical model of the role of RdCVF in the coexistence of rods and cones in a healthy eye
- Quantifying the metabolic contribution to photoreceptor death in retinitis pigmentosa via a mathematical model
- Scale-free neurodegeneration: cellular heterogeneity and the stretched exponential kinetics of cell death
- A mathematical model for photoreceptor interactions
- One-hit stochastic decline in a mechanochemical model of cytoskeleton-induced neuron death. III: Diffusion pulse death zones
- Tracing the progression of retinitis pigmentosa via photoreceptor interactions
- Bifurcation analysis of a photoreceptor interaction model for retinitis pigmentosa
- One-hit stochastic decline in a mechanochemical model of cytoskeleton-induced neuron death. I: Cell-fate arrival times
- One-hit stochastic decline in a mechanochemical model of cytoskeleton-induced neuron death. II: Transition state metastability
