Analysis of an HIV infection model incorporating latency age and infection age
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Publication:1714887
DOI10.3934/mbe.2018026zbMath1406.92627OpenAlexW2777870216WikidataQ58131290 ScholiaQ58131290MaRDI QIDQ1714887
Publication date: 1 February 2019
Published in: Mathematical Biosciences and Engineering (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.3934/mbe.2018026
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Cites Work
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- Global threshold dynamics in a five-dimensional virus model with cell-mediated, humoral immune responses and distributed delays
- Analysis of an age structured HIV infection model with virus-to-cell infection and cell-to-cell transmission
- A multi-strain virus model with infected cell age structure: application to HIV
- Global dynamics for a class of age-infection HIV models with nonlinear infection rate
- Global dynamics of a viral infection model with a latent period and Beddington-DeAngelis response
- Global dynamics of a cell mediated immunity in viral infection models with distributed delays
- Global stability of the endemic equilibrium in infinite dimension: Lyapunov functions and positive operators
- Global dynamics of an in-host viral model with intracellular delay
- Global stability for a HIV-1 infection model with cell-mediated immune response and intracellular delay
- Global stability of a five-dimensional model with immune responses and delay
- Semiflows generated by Lipschitz perturbations of non-densely defined operators
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Dynamical systems and evolution equations. Theory and applications
- To treat or not to treat: The case of tuberculosis
- Modeling HIV persistence, the latent reservoir, and viral blips
- An age-structured model of HIV infection that allows for variations in the production rate of viral particles and the death rate of productively infected cells
- Diseases with chronic stage in a population with varying size
- Global stability for an SEI epidemiological model with continuous age-structure in the exposed and infectious classes
- The stability analysis of a general viral infection model with distributed delays and multi-staged infected progression
- The dynamics of an SVIR epidemiological model with infection age: Table 1.
- Global Stability of a Nonlinear Viral Infection Model with Infinitely Distributed Intracellular Delays and CTL Immune Responses
- Lyapunov Functions and Global Stability for Age-Structured HIV Infection Model
- Global asymptotic stability for HIV-1 dynamics with two distributed delays
- Modeling HIV-1 Virus Dynamics with Both Virus-to-Cell Infection and Cell-to-Cell Transmission
- An HIV model with age-structured latently infected cells
- Mathematical analysis of an HIV latent infection model including both virus-to-cell infection and cell-to-cell transmission
- How May Infection-Age-Dependent Infectivity Affect the Dynamics of HIV/AIDS?
- A class of delayed viral models with saturation infection rate and immune response
- Impact of Intracellular Delays and Target-Cell Dynamics on In Vivo Viral Infections
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