Bridging blood cancers and inflammation: the reduced cancitis model
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Publication:1721804
DOI10.1016/j.jtbi.2019.01.001zbMath1406.92109OpenAlexW2908179281WikidataQ90923102 ScholiaQ90923102MaRDI QIDQ1721804
Niels Pallisgaard, Lasse Kjær, Trine A. Knudsen, Vibe Skov, Morten Andersen, Katrine O. Bangsgaard, Johanne Gudmand-Hoeyer, Zamra Sajid, Hans C. Hasselbalch, Rasmus Kristoffer Pedersen, Johnny Tom Ottesen
Publication date: 13 February 2019
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://rucforsk.ruc.dk/ws/files/64109874/Bridging_blood_cancer_and_inflammation_the_reduced_Cancitis_model.pdf
Related Items (2)
Global dynamics of healthy and cancer cells competing in the hematopoietic system ⋮ Dynamics of competing heterogeneous clones in blood cancers explains multiple observations -- a mathematical modeling approach
Cites Work
- Unnamed Item
- The resolution of inflammation: a mathematical model of neutrophil and macrophage interactions
- General model of inflammation
- Modeling immunotherapy of the tumor -- immune interaction
- Nonlinear dynamics of immunogenic tumors: Parameter estimation and global bifurcation analysis
- A mathematical model of tumor-immune evasion and siRNA treatment
- The dynamics of an optimally controlled tumor model: A case study
- Ordinary differential equation models for adoptive immunotherapy
- Stability analysis of a model of interaction between the immune system and cancer cells in chronic myelogenous leukemia
- Determining the role of inflammation in the selection of JAK2 mutant cells in myeloproliferative neoplasms
- A mathematical model for chronic myelogenous leukemia (CML) and T cell interaction
- Mixed immunotherapy and chemotherapy of tumors: modeling, applications and biological interpretations
- Patient specific modeling of the HPA axis related to clinical diagnosis of depression
- Integrated inflammatory stress (ITIS) model
- Mathematical modeling on helper T cells in a tumor immune system
- Optimal control of treatment in a mathematical model of chronic myelogenous leukemia
- Cancer self remission and tumor stability -- a stochastic approach
- Bifurcation patterns in generalized models for the dynamics of normal and leukemic stem cells with signaling
- Mathematical modelling of cytokine-mediated inflammation in rheumatoid arthritis
- Mathematical Modeling of Leukemogenesis and Cancer Stem Cell Dynamics
- Modeling tumor regrowth and immunotherapy
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