A hybrid multi-compartment model of granuloma formation and T cell priming in tuberculosis
From MaRDI portal
Publication:1783507
DOI10.1016/J.JTBI.2011.03.022zbMath1397.92348OpenAlexW2103406125WikidataQ37088339 ScholiaQ37088339MaRDI QIDQ1783507
Mohammed El-Kebir, Simeone Marino, Denise E. Kirschner
Publication date: 21 September 2018
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://ir.cwi.nl/pub/19157
Medical applications (general) (92C50) Cell biology (92C37) Cell movement (chemotaxis, etc.) (92C17)
Related Items (6)
Modelling the effects of environmental heterogeneity within the lung on the tuberculosis life-cycle ⋮ A low dimensional dynamical model of the initial pulmonary innate response to infection ⋮ On the stability of steady states in a granuloma model ⋮ To Sobol or not to Sobol? The effects of sampling schemes in systems biology applications ⋮ On the dynamics of radially symmetric granulomas ⋮ A population model capturing dynamics of tuberculosis granulomas predicts host infection outcomes
Uses Software
Cites Work
- A metapopulation model of granuloma formation in the lung during infection with Mycobacterium tuberculosis
- A comparison of random vs. chemotaxis-driven contacts of T cells with dendritic cells during repertoire scanning
- A methodology for performing global uncertainty and sensitivity analysis in systems biology
- Macrophage response to \textit{Mycobacterium tuberculosis} infection
- Identifying control mechanisms of granuloma formation during \textit{M. tuberculosis} infection using an agent-based model
- The human immune response to \textit{Mycobacterium tuberculosis} in lung and lymph node
- The role of delays in innate and adaptive immunity to intracellular bacterial infection
- Sensitivity and Uncertainty Analysis of Complex Models of Disease Transmission: An HIV Model, as an Example
This page was built for publication: A hybrid multi-compartment model of granuloma formation and T cell priming in tuberculosis
