The timing of TNF and IFN-\(\gamma\) signaling affects macrophage activation strategies during \textit{Mycobacterium tuberculosis} infection

DOI10.1016/j.jtbi.2008.01.010zbMath1398.92248OpenAlexW2072878147WikidataQ40087615 ScholiaQ40087615MaRDI QIDQ1794413

Yanyan Li

Publication date: 15 October 2018

Published in: Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1016/j.jtbi.2008.01.010

zbMATH Keywords

response timefeedbackmathematical modelimmune systemcytokine

Mathematics Subject Classification ID

Epidemiology (92D30) Biochemistry, molecular biology (92C40)

Related Items (3)

Understanding PGE2, LXA4 and LTB4 balance during \textit{Mycobacterium tuberculosis} infection through mathematical model ⋮ Mathematical model of mycobacterium-host interaction describes physiology of persistence ⋮ A mathematical representation of the development of \textit{Mycobacterium tuberculosis} active, latent and dormant stages

Cites Work

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• Alternative designs for a genetic switch: Analysis of switching times using the piecewise power-law representation
• Requirement for multiple activation signals by anti-inflammatory feedback in macrophages
• Design principles for elementary gene circuits: Elements, methods, and examples
• A Comparison of Three Methods for Selecting Values of Input Variables in the Analysis of Output from a Computer Code
• Sensitivity and Uncertainty Analysis of Complex Models of Disease Transmission: An HIV Model, as an Example

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