Resistance to protease inhibitors in a model of HIV-1 infection with impulsive drug effects
From MaRDI portal
Publication:2440890
DOI10.1007/s11538-013-9903-9zbMath1283.92050OpenAlexW2166026801WikidataQ42257557 ScholiaQ42257557MaRDI QIDQ2440890
Rachelle E. Miron, Robert J. Smith?
Publication date: 20 March 2014
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s11538-013-9903-9
Ordinary differential equations with impulses (34A37) Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60) Control problems involving ordinary differential equations (34H99)
Related Items (5)
Global dynamics of a state-dependent feedback control system ⋮ Nonlinear multi-term impulsive fractional \(q\)-difference equations with closed boundary conditions ⋮ A mathematical model on acquired immunodeficiency syndrome ⋮ MODELING MONOCYTE-DERIVED DENDRITIC CELLS AS A THERAPEUTIC VACCINE AGAINST HIV ⋮ On periodic solutions of delay differential equations with impulses
Cites Work
- Distinct effects of protease and reverse transcriptase inhibition in an immunological model of HIV-1 infection with impulsive drug effects
- Drug resistance in an immunological model of HIV-1 infection with impulsive drug effects
- The role of backward mutations on the within-host dynamics of HIV-1
- Threshold virus dynamics with impulsive antiretroviral drug effects
- On the definition and the computation of the basic reproduction ratio \(R_ 0\) in models for infectious diseases in heterogeneous populations
- Can the viral reservoir of latently infected \(CD4^{+}\) T cells be eradicated with antiretroviral HIV drugs?
- Mechanism-based model of the pharmacokinetics of enfuvirtide, an HIV fusion inhibitor
- A viral load-based cellular automata approach to modeling HIV dynamics and drug treatment
- Dynamics of HIV infection of CD4\(^ +\) T cells
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Optimal drug treatment regimens for HIV depend on adherence
- Modeling within-host HIV-1 dynamics and the evolution of drug resistance: trade-offs between viral enzyme function and drug susceptibility
- Emergence of HIV-1 drug resistance during antiretroviral treatment
- Predicting the potential impact of a cytotoxic T-lymphocyte HIV vaccine: How often should you vaccinate and how strong should the vaccine be?
- Modelling the effects of adherence to the HIV fusion inhibitor enfuvirtide
- Hierarchical Bayesian inference for HIV dynamic differential equation models incorporating multiple treatment factors
- Drug concentration heterogeneity facilitates the evolution of drug resistance
- Sensitivity and Uncertainty Analysis of Complex Models of Disease Transmission: An HIV Model, as an Example
- Unnamed Item
- Unnamed Item
- Unnamed Item
- Unnamed Item
- Unnamed Item
This page was built for publication: Resistance to protease inhibitors in a model of HIV-1 infection with impulsive drug effects