Modeling the role of TGF-\(\beta\) in regulation of the Th17 phenotype in the LPS-driven immune system

DOI10.1007/s11538-014-9946-6zbMath1297.92019OpenAlexW2080686782WikidataQ51103663 ScholiaQ51103663MaRDI QIDQ458719

Seongwon Lee, Hyung-Ju Hwang, Yang-Jin Kim

Publication date: 8 October 2014

Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1007/s11538-014-9946-6

zbMATH Keywords

lipopolysaccharidephenotypic switchesTGF-\(\beta\)Th17 phenotype

Mathematics Subject Classification ID

Physiology (general) (92C30)

Related Items (4)

Spatio-temporal models of lymphangiogenesis in wound healing ⋮ Mathematical modeling and its analysis for instability of the immune system induced by chemotaxis ⋮ Bifurcations and Multistability in a Model of Cytokine-Mediated Autoimmunity ⋮ Hopf bifurcation in a model of TGF-\(\beta\) in regulation of the Th17 phenotype

Uses Software

• Matlab

Cites Work

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• Mathematical modeling of allergy and specific immunotherapy: Th1-Th2-Treg interactions
• Multi-scale models of cell and tissue dynamics
• The role of the microenvironment in tumor invasion
• A HYBRID MODEL FOR TUMOR SPHEROID GROWTH IN VITRO I: THEORETICAL DEVELOPMENT AND EARLY RESULTS
• Unnamed Item

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