BISTABILITY ANALYSIS OF AN HIV MODEL WITH IMMUNE RESPONSE
From MaRDI portal
Publication:4684140
DOI10.1142/S021833901740006XzbMath1397.92410OpenAlexW2781457135MaRDI QIDQ4684140
No author found.
Publication date: 27 September 2018
Published in: Journal of Biological Systems (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1142/s021833901740006x
Bifurcation theory for ordinary differential equations (34C23) Global stability of solutions to ordinary differential equations (34D23) Medical epidemiology (92C60)
Related Items (10)
Interlocked feedback loops balance the adaptive immune response ⋮ Bifurcations and Bistability of an Age-Structured Viral Infection Model with a Nonmonotonic Immune Response ⋮ Unnamed Item ⋮ Complex dynamics for an immunosuppressive infection model with virus stimulation delay and nonlinear immune expansion ⋮ HIV infection dynamics and viral rebound: modeling results from humanized mice ⋮ Qualitative analysis of peer influence effects on testing of infectious disease model ⋮ Analysis of an HIV model with post-treatment control ⋮ An optimal scheme to boost immunity and suppress viruses for HIV by combining a phased immunotherapy with the sustaining antiviral therapy ⋮ Analysis of an HIV model with immune responses and cell-to-cell transmission ⋮ Monotonic and nonmonotonic immune responses in viral infection systems
Cites Work
- Unnamed Item
- Global analysis for delay virus dynamics model with Beddington-DeAngelis functional response
- A delayed HIV-1 infection model with Beddington-DeAngelis functional response
- Dynamics analysis of a delayed viral infection model with immune impairment
- Complex dynamic behavior in a viral model with delayed immune response
- Global stability of an age-structured virus dynamics model with Beddington-DeAngelis infection function
- A simple model of pathogen-immune dynamics including specific and non-specific immunity
- Stability and Hopf bifurcation for a viral infection model with delayed non-lytic immune response
- Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
- Global properties for virus dynamics model with Beddington-DeAngelis functional response
- A delay-differential equation model of HIV infection of \(\text{CD}4^+\) T-cells
- Mathematical analysis of delay differential equation models of HIV-1 infection
- Mathematical modeling of viral kinetics under immune control during primary HIV-1 infection
- Modeling HIV persistence, the latent reservoir, and viral blips
- Immune impairment in HIV infection: existence of risky and immunodeficiency thresholds
- A methodology for performing global uncertainty and sensitivity analysis in systems biology
- Dynamics analysis of a delayed viral infection model with logistic growth and immune impairment
- Predicting the HIV/AIDS epidemic and measuring the effect of mobility in mainland China
- A chronic viral infection model with immune impairment
- Piecewise virus-immune dynamic model with HIV-1 RNA-guided therapy
- Virus Dynamics: A Global Analysis
- Sensitivity and Uncertainty Analysis of Complex Models of Disease Transmission: An HIV Model, as an Example
- Persistence under Relaxed Point-Dissipativity (with Application to an Endemic Model)
This page was built for publication: BISTABILITY ANALYSIS OF AN HIV MODEL WITH IMMUNE RESPONSE
