Accounting for multi-delay effects in an HIV-1 infection model with saturated infection rate, recovery and proliferation of host cells
From MaRDI portal
Publication:5050553
DOI10.11145/j.biomath.2020.12.297OpenAlexW3115148312MaRDI QIDQ5050553
Robert Hakl, Debadatta Adak, Nandadulal Bairagi
Publication date: 17 November 2022
Published in: BIOMATH (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.11145/j.biomath.2020.12.297
Epidemiology (92D30) Stability theory of functional-differential equations (34K20) Bifurcation theory of functional-differential equations (34K18)
Related Items (2)
Accounting for multi-delay effects in an HIV-1 infection model with saturated infection rate, recovery and proliferation of host cells ⋮ Bifurcation analysis of HIV infection model with cell-to-cell transmission and non-cytolytic cure
Cites Work
- Unnamed Item
- Unnamed Item
- Unnamed Item
- Unnamed Item
- Unnamed Item
- A model of HIV-1 infection with two time delays: mathematical analysis and comparison with patient data
- Global stability of an HIV-1 infection model with saturation infection and intracellular delay
- Global stability for an HIV-1 infection model including an eclipse stage of infected cells
- Global dynamics of an in-host viral model with intracellular delay
- A differential equation model of HIV infection of CD4\(^{+}\) T-cells with delay
- A delay-differential equation model of HIV infection of \(\text{CD}4^+\) T-cells
- Global analysis of HIV-1 dynamics with Hill type infection rate and intracellular delay
- Dynamics of HIV infection of CD4\(^ +\) T cells
- Optimal HIV treatment by maximising immune response
- A model of HIV-1 pathogenesis that includes an intracellular delay
- Complex patterns of viral load decay under antiretroviral therapy: influence of pharmacokinetics and intracellular delay
- Numerical study for a novel variable-order multiple time delay awareness programs mathematical model
- Modeling within-host HIV-1 dynamics and the evolution of drug resistance: trade-offs between viral enzyme function and drug susceptibility
- Dynamics analysis and simulation of a modified HIV infection model with a saturated infection rate
- Oscillatory viral dynamics in a delayed HIV pathogenesis model
- Analysis and computation of multi-pathways and multi-delays HIV-1 infection model
- Global stability and periodic solution of a model for HIV infection of CD4\(^{+}\) T cells
- A primary infection model for HIV and immune response with two discrete time delays
- A differential equation model of HIV infection of CD4\(^+\) \(T\)-cells with cure rate
- Mathematical analysis of the global dynamics of a model for HIV infection of CD4\(^{+}\) T cells
- Asymptotic properties of a HIV-1 infection model with time delay
- Modeling the dynamics of HIV and \(CD4^{+}\)T cells during primary infection
- Global Stability of a Nonlinear Viral Infection Model with Infinitely Distributed Intracellular Delays and CTL Immune Responses
- Persistence in Infinite-Dimensional Systems
- Analysis of HIV models with two time delays
- Systems of Differential Equations That are Competitive or Cooperative. IV: Structural Stability in Three-Dimensional Systems
- Impact of delays in cell infection and virus production on HIV-1 dynamics
- Uniformly Persistent Systems
- Virus Dynamics: A Global Analysis
- Sensitivity and Uncertainty Analysis of Complex Models of Disease Transmission: An HIV Model, as an Example
- Accounting for multi-delay effects in an HIV-1 infection model with saturated infection rate, recovery and proliferation of host cells
- Bifurcation analysis of a multidelayed HIV model in presence of immune response and understanding of in‐host viral dynamics
This page was built for publication: Accounting for multi-delay effects in an HIV-1 infection model with saturated infection rate, recovery and proliferation of host cells
