A dual negative regulation model of Toll-like receptor 4 signaling for endotoxin preconditioning in human endotoxemia
DOI10.1016/J.MBS.2011.05.005zbMath1218.92029OpenAlexW2107338366WikidataQ51563471 ScholiaQ51563471MaRDI QIDQ554488
Stephen F. Lowry, Ioannis P. Androulakis, Steven E. Calvano, Qian Yang
Publication date: 4 August 2011
Published in: Mathematical Biosciences (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.mbs.2011.05.005
Probabilistic models, generic numerical methods in probability and statistics (65C20) Biomedical imaging and signal processing (92C55) Medical applications (general) (92C50) Biochemistry, molecular biology (92C40) Physiology (general) (92C30)
Uses Software
Cites Work
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- Mechanistic simulations of inflammation: current state and future prospects
- A simple mathematical model of signaling resulting from the binding of lipopolysaccharide with toll-like receptor 4 demonstrates inherent preconditioning behavior
- Modeling endotoxin-induced systemic inflammation using an indirect response approach
- A model of TLR4 signaling and tolerance using a qualitative, particle-event-based method: Introduction of spatially configured stochastic reaction chambers (SCSRC)
- A reduced mathematical model of the acute inflammatory response. II: Capturing scenarios of repeated endotoxin administration
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