Modeling identifies variability in SARS-CoV-2 uptake and eclipse phase by infected cells as principal drivers of extreme variability in nasal viral load in the 48 h post infection

DOI10.1016/J.JTBI.2023.111470zbMath1518.92047MaRDI QIDQ6174208

Ronit Freeman, Jason Pearson, Richard C. Boucher, Samuel K. Lai, Alex W. J. Chen, M. Gregory Forest, Timothy Wessler, Raymond Pickles

Publication date: 14 July 2023

Published in: Journal of Theoretical Biology (Search for Journal in Brave)

zbMATH Keywords

viral loadmechanistic modelingSARS-CoV-2nasal infection

Mathematics Subject Classification ID

Pathology, pathophysiology (92C32)

Cites Work

• A hybrid discrete-continuum model of immune responses to SARS-CoV-2 infection in the lung alveolar region, with a focus on interferon induced innate response

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