Studying HIV latency by modeling the interaction between HIV proteins and the innate immune response

DOI10.1016/j.jtbi.2014.06.025zbMath1343.92274OpenAlexW1983065235WikidataQ42208000 ScholiaQ42208000MaRDI QIDQ739663

Luis U. Aguilera, Jesús Rodríguez-González

Publication date: 19 August 2016

Published in: Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1016/j.jtbi.2014.06.025

zbMATH Keywords

bistabilityinnate immune systemintracellular dynamicsrestriction factortat circuit

Mathematics Subject Classification ID

Medical applications (general) (92C50) Medical epidemiology (92C60)

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Cites Work

• Asymmetric division of activated latently infected cells may explain the decay kinetics of the HIV-1 latent reservoir and intermittent viral blips
• On a resource based ecological competition model with interference
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• Brownian motion in a field of force and the diffusion model of chemical reactions